Originally from: mark purdey
Hi folks
Since BSE and other TSEs first hit the headlines in the 1980s, a whole history book of ' unexpected' results has arisen at every juncture of this unsavoury man made crisis. Each one has seriously rocked the boat of credibility surrounding the official infectious theory.
But in the ideal world, any impartial official body which holds the best interests of public / animal health at heart, would simply return to the drawing board whenever one of these epidemiological hiccoughs emerged. But with MAFF/DEFRA at the helm, this kind of responsible lateral approach has never been taken. They merely choose to squeeze more blood out of the sinking stone, and move the goalposts – often to ridiculous extremes – as a last ditch attempt to keep their die hard, dumbo theory afloat.
This latest challenge involves the surprise development of clinical scrapie amongst a supposedly totally scrapie-resistant breed of sheep, which , of course, makes a total mockery of the effectiveness of their National Scrapie Plan – costing millions of
pounds of taxpayers money,. thousands of healthy animals' lives, and hundreds of traditional sheep flocks that have been diligently bred over the centuries.
For what it is worth, I have spent the last fifteen years travelling the world carrying out environmental analyses of all well renowned clusters of TSE. My work has identified a common toxic denominator that is present in everyone of these environments, and the less sensitive data that I have collected has been published in peer reviewed academic journals.
Within most of the cluster areas, It is not unusual to observe a high concentration of cases of both genetically induced forms of TSE as well as the sporadically induced TSEs – suggesting that both forms of this rare disease are caused by exposure to the same common factor that is exclusive to these cluster environments.
Since this environmental factor can invoke disease – either via a frame shift mutation of the genetic material, or via a straight forward disruption of prion protein function -then surely any breed of sheep is, to varying degrees, going to be susceptible to this kind of environmentally induced mutation.
Unless the government faces reality and seeks to prevent TSEs by dealing with the root of the
TSE problem (ie; environmental pollution) as opposed to sweeping its effects under the carpet (by a sustained annihilation of the victims themselves), then the Treasury is going to have to keep on coughing up millions of our taxes
– purely to appease the insecure egos of a handful of misguided and wholly unintelligent government scientists.
Best







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